Know the possible pathogenesis of HUS and TTP
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10.12.08
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Probably involves glomerular endothelial cell injury with subsequent fibrin deposition and thrombosis
Decreased endothelial cell production of prostaglandin I2 and NO
Release of endothelin
Activation of endothelial cells, increasing their adhesiveness to leukocytes, contributing to thrombosis
Category: Pathology Notes
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